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Dinitrophenol, an agent that acts in a manner similar to ouabain by accumulating sodium into the renal tubule, was administered intraperitoneally to produce ascites in rats. Urine osmolalities of control and ascitic rats indicated that more than half of ascitic urine was concentrated and in the osmotically equivalent state to that of serum. Measurements of blood gases in animals with induced ascites showed that Pao2 was markedly decreased and Pco2 markedly increased. The ascites contained a large amount of sodium when generated in a high sodium diet. After the administration of dinitrophenol the urine osmolality decreased and sodium excretion increased markedly. A satisfactory decrease of ascites volume occurred within 24 h after the intraperitoneal administration of dinitrophenol. It was suggested that the ouabain-like compound dinitrophenol was useful in the treatment of ascites. Case: 18-40715
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